Ginger root is an incredible addition to almost all styles of cuisine, and it’s surprisingly easy to grow. There are a few different methods you can use, all of which work well. Some people just cut pieces of ginger, making sure a few eyes are on each piece and plant directly. In this video, we are going to be growing ginger from the store by starting it off in bowls of water. Once new shoots and roots start developing from the growth nodes, we can either plant them directly in the soil, or snap them off and plant those pieces directly into the soil. Growing ginger in containers is easy, too – it loves a nutrient-rich, moist soil and plenty of sun. So long as you give it those two things, it’ll grow vigorously and will be ready to harvest about ~4 months from planting. In a perfect world, you’d plant ginger in the spring for a fall harvest, but depending on your climate you can get away with either a shorter or longer growing season. LEARN MORE Epic Gardening is much more than a YouTube channel. I have a website with 300+ gardening tutorials as well as a podcast where I release daily gardening tips in five minutes or less. There’s also a Facebook group with over 1,500 other gardeners sharing their tips. → Website: http://www.epicgardening.com/blog → Podcast: https://apple.co/2nkftuk → FB Group: https://www.facebook.com/groups/epicg… DONATE If you like my videos, articles, or podcast episodes, please consider supporting on Patreon. For rewards, I’ll answer gardening questions and make videos! → https://www.patreon.com/epicgardening SOCIAL MEDIA → Steemit: https://steemit.com/@halcyondaze → Instagram: https://www.instagram.com/epicgardening/ → Pinterest: https://www.pinterest.com/epicgardening → Facebook: http://www.facebook.com/epicgardening → Twitter: http://www.twitter.com/epicgardening
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In this broadcast I touch on weird dreams, Ex lovers that wont let go as well as the ones that never really had a grip to begin with. Updates on Blue Oyster projects. Perfect timing
Quotes of the day
When you see old lady run … no ask whats the matter just run too
Pig and mud go together
People often agree in words but no in judgement
Two people in accord are stronger than 8 that disagree.
Sure, you can buy turmeric powder from the spice department to whip up your own golden milk, turmeric lattes, turmeric smoothies, or turmeric tea, but aficionados swear by fresh turmeric for the best flavor and possible health benefits. And while you can find the fresh stuff in health food stores and even mainstream grocery stores, it isn’t cheap.
Luckily turmeric is easy to grow if you have a sunny spot to put a large pot or planter. Give it what it likes and it will grow like a weed and reward you with attractive tropical foliage and a generous harvest of fresh turmeric.
Turmeric (Curcuma longa) is a tropical plant in the same family as ginger. Not a dainty plant, turmeric has large green leaves and grows 3 or more feet tall. As the plant matures each stem sends up a spike of greenish-white and occasionally pink flowers. Like ginger, turmeric thrives in warm, humid conditions and well-drained, neutral soil.
In most parts of the U.S. turmeric will produce best if you plant it indoors in the late winter. Depending on your indoor and outdoor space you can either keep it inside as a houseplant all summer or move it outside once all chance of frost is past and the weather is warm enough to put out your pepper and eggplant seedlings. And if you live in Zones 8-11, you can grow it completely outdoors.
Turmeric takes seven to 10 months from planting to harvest. To figure out when you should plant, count back 10 months from when you usually get your first frost in the fall. My first frost is around mid-October, so I’d start my turmeric between mid-December and mid-March. If your growing season is longer, or you have a large and sunny indoor space to grow it, your timing is less critical, but you’re still likely to get the best results from planting in late winter through spring.
Turmeric is grown from rhizomes, fleshy root-like structures. My local supermarket and health food store both have fresh rhizomes for sale in the winter. Asian or Indian groceries are also likely to stock it, or may be able to order some for you. If you can’t find any locally, Jung Seed sells small potted plants, or you can buy fresh turmeric rhizomes from a number of sellers on Amazon or eBay. (Choose a seller in the U.S. to avoid possible customs issues). Select plump rhizomes with as many bumps (buds) along the sides as possible.
You will need a 14- to 18-inch pot or planter for each 6 to 8 inches of rhizome, and enough potting soil to fill it. But to start, it’s more practical to sprout your rhizomes in smaller containers and then transplant them into the larger containers once they have a few leaves and are growing well. Here’s how:
1. Cut your rhizomes into sections, with two or three buds on each section.
2. Fill 3-inch pots halfway with a good potting soil.
3. Lay the rhizome sections flat on the soil, and cover with more potting soil.
4. Water well and slip the pots into clear plastic bags.
5. Place the pots or clamshells in the warmest place you can find (86 to 95 degrees is ideal). Sprouting at lower temperatures will be very slow and your rhizomes may even rot rather than sprout. No toasty location? You can make a great germination chamber with a heating pad or a small desk lamp, a picnic cooler, and a thermometer. Or you can buy a small germination chamber for home use. Light or no light is fine at this stage.
Check on your pots every few days and once the sprouts start to emerge, move the pots to a windowsill or under a grow light. Unless your house is really warm (optimal growing temperature at this stage is 77 to 86 degrees) you will want to put them on a heat mat set to the low 80s. As the plants outgrow their plastic covers, remove them.
Once you open the mini greenhouses you will need to start watering your turmeric as needed; keep the soil moist, but not soggy, and mist the leaves once or twice a day with water to keep the humidity up. Allowing the soil to dry out at any point will reduce your final harvest.
When your plants are 6 to 8 inches tall, carefully transplant them into larger pots (either the final ones or an intermediate size) full of potting soil. Begin turning the heat mat down several degrees each week until you hit 70 degrees. At this point, you can remove the heat mat as long as your indoor temperature averages at about 68 degrees.
Otherwise, continue using the heat mat. Plants in intermediate-sized pots are ready to go in their final pots or planters when they become top-heavy or start sending up more shoots.
Move your turmeric outside once all chance of frost is past, when the forecast shows only warm nights ahead. Provide partial shade for the first few days to keep tender leaves from getting sunburned. Continue to water as needed during the summer and fall to keep the soil moist but not soggy. Feed your growing plants by watering every couple of weeks with compost tea or applying a recommended for potatoes or root crops.
Your turmeric is ready to harvest when the leaves and stem start to turn brown and dry, about seven to 10 months after planting. Tip out the plants, soil and all, and shake the soil off your fresh turmeric. Cut the stems off an inch or so above the mass of rhizomes and wash the rhizomes well.
Rhizomes will stay fresh in the fridge for up to six months in an airtight bag or container; toss them in the freezer to save them for longer. Be sure to set a few of the largest aside for replanting!
You can also make your own turmeric powder. Place the freshly cleaned rhizomes in a pot and cover them with water, bring them to a boil, and simmer until you can easily pierce them with a fork (depending on their size, this may take 45 to 60 minutes or longer).
Drain the cooked rhizomes, rub the skin off with your fingers (optional), and dry them in the sun or a food dehydrator set at 140 degrees until they are brittle and snap cleanly when you try to bend them. Grind dried rhizomes in a spice mill, coffee grinder, or mortar and pestle to make turmeric powder for cooking. Pro tip: You may want to wear gloves when handling turmeric rhizomes as they will turn your fingers a bright orange that won’t wash off.
Enjoy your tasty and healthful harvest!
Source – Good House Keeping
Glutamate is the most abundant neurotransmitter in our brain and central nervous system (CNS). It is involved in virtually every major excitatory brain function. While excitatory has a very specific meaning in neuroscience, in general terms, an excitatory neurotransmitter increases the likelihood that the neuron it acts upon will have an action potential (also called a nerve impulse).1 When an action potential occurs the nerve is said to fire, with fire, in this case, being somewhat akin to the completion of an electric circuit that occurs when a light switch is turned on. The result of neurons firing is that a message can be spread throughout the neural circuit. It is estimated that well over half of all synapses in the brain release glutamate, making it the dominant neurotransmitter used for neural circuit communication.
Glutamate is also a metabolic precursor for another neurotransmitter called GABA (gamma-aminobutyric acid). GABA is the main inhibitory neurotransmitter in the central nervous system. Inhibitory neurotransmitters are essentially the flip-side of the coin—they decrease the likelihood that the neuron they act upon will fire.1
In the brain, groups of neurons (nerve cells) form neural circuits to carry out specific small-scale functions (e.g., formation and retrieval of memory). These neural circuits interconnect with each other to form large-scale brain networks, which carry out more complex functions (e.g., hearing, vision, movement). In order to get the individual nerve cells to work together across these networks some type of communication between them is needed and one way it is accomplished is by chemical messenger molecules called neurotransmitters. Glutamate plays a prominent role in neural circuits involved with synaptic plasticity—the ability for strengthening or weakening of signaling between neurons over time to shape learning and memory. It’s a major player in the subset of plasticity called long-term potentiation (LTP).
“The brain doesn’t grow new neurons to store memories. It strengthens connections between existing neurons. This process is called long-term potentiation (LTP).”
Because of these and other roles, the glutamatergic system is paramount for fast signaling and information processing in neuronal networks. Glutamate signaling is critical in brain regions, including the cortex and hippocampus, which are fundamental for cognitive function. Glutamate receptors are widely expressed throughout the CNS, not only in neurons, but also in glial cells.
[Note: Glial cells (or neuroglia or simply glia) are non-neuronal brain cells that provide support and protection for neurons.]
Because it is the main molecule promoting neuronal excitation, glutamate is the principal mediator of cognition, emotions, sensory information, and motor coordination, and is linked to the activity of most other neurotransmitter systems.2 But glutamate is not a “more is better” molecule. Glutamatergic communication requires the right concentrations of glutamate be released in the right places for only small amounts of time. Less than this results in poor communication. More than this can be neurotoxic and can damage neurons and neural networks.
Glutamate signaling is an example of what’s sometimes referred to as the “Goldilocks Principle.” In the fairy tale story, Goldilocks tastes three different bowls of porridge. The first is too cold; the second is too hot, and the third is just the right temperature. This concept of a “just the right amount” has widespread application, including in cognitive science.
In cognitive science, this principle can refer to a process where the same neurotransmitter (or medication) can have both antagonist (inhibitory) and agonist (excitatory) properties. It can also apply to situations where too little or too much stimulation by the same signaling molecule is linked to sub-optimal performance, but some middle ground amount produces healthy responses.
When thinking about things that follow the Goldilocks Principle it’s important to avoid black-white or good-bad thinking. The key thing to focus on is that there’s a just right amount, often a range, where the best results are produced.
“Glutamate follows the Goldilocks Principle. Too little glutamate excitation can result in difficulty concentrating or mental exhaustion. But too much can result in excitotoxicity, which can damage nerve cells (neurons).”
Neurotransmitters have several characteristics in common. The first is that they are synthesized (i.e., made or created) in neurons. After that, they are moved into areas near the end of neurons (synaptic vesicles near the terminal end of nerve cells) where they are stored until needed. This occurs in preparation for signaling, which involves release of the neurotransmitter from the message-sending neuron into the space between neurons (synaptic cleft), so it can activate (i.e., bind to) receptors on message-receiving neurons. After this signal is sent, the space between neurons is cleaned up, so it can be made ready for the next time a message needs to be sent. This is achieved by absorbing the neurotransmitter into a cell so it can be reused (recycling), and/or by degrading (breaking down and inactivating) the neurotransmitter in the space outside cells. Let’s explore how these occur with glutamate.
Glutamate does not cross the blood-brain barrier and must be synthesized in neurons from building block molecules (i.e., precursors) that can get into the brain. In the brain, glutamine is the fundamental building block for glutamate. The most prevalent biosynthetic pathway synthesizes glutamate from glutamine using an enzyme called glutaminase.2
[Note: Enzymes are catalysts used to produce specific biochemical reactions: They usually have names that end in “ase.” Coenzymes are parts of certain enzymes. Many coenzymes are derived from vitamins.]
Glutamine is the most abundant of the twenty amino acids the body uses to build proteins. It can be produced in the body (so is categorized as non-essential). Most glutamine is made and stored in muscle. Under certain circumstances, such as severe stress, the body can require more than it can make. This has led many scientists to consider glutamine as being a conditionally essential amino acid. It is one of the few amino acids that can directly cross the blood–brain barrier, so the glutamine pool in muscle can be used to support the brain.
“The blood-brain barrier acts a bit like a doorman, choosing what goes in (like nutrients) and out (such as metabolic waste products) of the brain. It also protects the brain against the entry of potentially harmful things (like bacteria).”
Glutamate can also be produced from glucose through a metabolic pathway that begins with the conversion of glucose to pyruvate (a process called glycolysis). Pyruvate then ethers the tricarboxylic acid (TCA) cycle (also called the Krebs cycle or citric acid cycle). The TCA cycle forms multiple important intermediates. One of these intermediates is α-ketoglutarate (α-KG). α-KG can be used to produce glutamate. An enzyme called glutamate dehydrogenase, which uses vitamin B3 (NAD+) as a coenzyme, is responsible for this reaction. This same enzyme can reconvert glutamate back into α-KG. Because of this enzyme, glutamate and α-KG can be continuously converted into each other.2 This dynamic equilibrium is a key intersection between anabolic and catabolic pathways and allows the body to shift resources in whichever direction is required.
[Note: Anabolic pathways construct molecules from smaller units. Catabolic pathways break molecules down into smaller units.]
Neurotransmitters, including glutamate, convey information from one neuron (message sender) to other “target” neurons (message recipients) within neural circuits. After synthesis, glutamate is transported into synaptic vesicles by vesicular glutamate transporters. This transport (and storage) occurs in the message-sending neuron in anticipation of needing to send glutamate messages in the future. Glutamate is stored in these vesicles until a nerve impulse triggers the release of glutamate into the synaptic cleft (i.e.,the space between neurons) and starts a receptor-mediated signaling process.3
“It’s estimated that about 99.99% of all the glutamate in the brain is stored inside cells (intracellular). Intracellular glutamate is inactive. It’s only the glutamate in the extracellular space between cells that causes excitation.”
Neurons with glutamate receptor proteins (i.e., glutamate receivers) respond to glutamate in the synaptic cleft. There are two general types of glutamate receptors. One type are called ionotropic receptors: Glutamate binding to these receptors allows the entry of ions (i.e., electrically charged minerals such as sodium or calcium) into the cell. There are three classes of ionotropic glutamate receptors: (1) N-methyl-D-aspartate (NMDA), (2) α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA), and (3) kainate receptors.
The second type of receptors are linked to molecules that will activate intracellular signaling pathways subsequent to glutamate binding. These are called G protein-coupled or metabotropic receptors. Metabotropic glutamate receptors (mGluR) modulate synaptic transmission (i.e. neuronal communication) by regulating the activity of a wide variety of ion channels, including ionotropic glutamate receptors, as well as receptors for other neurotransmitters.1,4
The next phase of neurotransmission is cleanup. Signaling is based on relative changes, not absolute amounts. In a quiet room the human ear might detect a whisper. In a loud nightclub it might not hear a shout. Neurotransmitters work on a similar principle. Short bursts of glutamate produce responses. But, for the best response to occur with the smallest amount of glutamate, the space between neurons needs to be the equivalent of a quiet room. Glutamate also follows the Goldilocks principle—too little and signaling within neural networks is subpar, but too much can be neurotoxic. For these reasons, the glutamate in the extracellular space between neurons needs to be continuously removed.
Neurotransmitter cleanup is commonly a combination of (1) transporting some of the neurotransmitter back into cells, and (2) inactivating the neurotransmitter that’s left floating in the space between cells. While the first of these processes applies to glutamate, there is no enzymatic inactivation system for glutamate in the extracellular space. This means that glutamate can interact with its receptors continuously until it diffuses away or is taken up by cellular transporters for reuse/recycling.
“Because there are no enzyme systems in the spaces between nerve cells to inactivate glutamate, it’s important to support nerve cells against excessive glutamate excitation. Astrocytes provide part of this support.”
Some glutamate can be taken up into neurons. This is done by excitatory amino acid transporters (i.e., glutamate transporters), but much of the released glutamate is taken up by a type of glial cell called astroglia or astrocytes. Astroglia surround synapses and play important roles in areas including nervous system repair, metabolic support of neurons, and neurotransmitter cleanup. The combination of neurons and supporting astroglia are responsible for emptying the synaptic cleft of glutamate to turn off the signal and reset the system for generation and propagation of the next glutamate signal. In this cleanup role, astroglia act to protect neurons from glutamate excitotoxicity.
Source – NeuroHacker
Researchers studying the loss of ego commonly experienced while tripping on psychedelics such as magic mushrooms have discovered that a key neurotransmitter may be linked to the phenomenon. A report on the research by scientists at Maastricht University in the Netherlands was published last month in the journal Neuropsychopharmacology.
Quite often, those who use psychedelic drugs such as psilocybin mushrooms and LSD experience a change in the perception of self and one’s connection to the larger world. Known as ego-dissolution, ego-disintegration, or ego-loss, the experience can result in a reduced state of self-awareness and a disruption of the boundaries of self and the world, leading to an increased feeling of unity with the environment. For some, the experience of ego-dissolution is a positive one, or a good trip, while others may have a negative, sometimes terrifying, bad trip.
Previous research has shown that ego and self-awareness may be related to levels of glutamate, the brain’s most abundant neurotransmitter. To test the theory, the team of researchers at Maastricht University designed a double-blind and placebo-controlled experiment to study the effect that psilocybin had on the glutamate levels of 60 healthy volunteers. Brain activity of the test subjects was monitored using magnetic resonance imaging (MRI).
The researchers discovered that psilocybin led to increased levels of glutamate in an area of the brain known as the prefrontal cortex, which is thought to be responsible for planning complex behavior, personality expression, decision-making, and moderating social behavior. They also recorded lower levels of glutamate in the hippocampus, an area of the brain that has been linked to the formation of memories and one’s sense of self-esteem.
The researchers also noted that the higher levels of glutamate in the prefrontal cortex caused by taking psilocybin were associated with a good trip, while the lower levels of glutamate in the hippocampus were linked to experiencing a bad trip.
“Whereas changes in [cortical] glutamate were found to be the strongest predictor of negatively experienced ego dissolution, changes in hippocampal glutamate were found to be the strongest predictor of positively experienced ego dissolution,” the researchers wrote.
While it’s yet not clear if glutamate is actually related to the experience of ego-dissolution, other studies have suggested that psychedelics may decouple different regions of the brain.
“Our data add to this hypothesis, suggesting that modulations of hippocampal glutamate in particular may be a key mediator in the decoupling underlying feelings of (positive) ego dissolution,” wrote the researchers.
Although more research is needed, the discovery of psilocybin’s effect on glutamate levels could help explain how the drug can be used therapeutically for a variety of mental health conditions including depression and severe anxiety.
“Such findings provide further insights into the underlying neurobiological mechanisms of the psychedelic state, and importantly, provide a neurochemical basis for how these substances alter individuals’ sense of self, and may be giving rise to therapeutic effects witnessed in ongoing clinical trials,” the authors wrote.
Source – HighTimes
PUBLISHED: JUL 9, 2020 8:24 AM
Alack of representation in Egypt is why Durand Reeves founded Fugee Run – a running club that is creating a safe mental and physical health space for people of color living in Egypt.
“Visibly seeing beautiful Black Africans all over the country but not seeing these same faces represented in the spaces that I was dealing in, was not something I could accept,” Reeves said. “Searching for opportunities to grow my business, while elevating the African communities here, I saw a void in the running space.”
The group started with just 12 people and has since grown to more than 120 runners that meet early on Mondays, Wednesdays, and Fridays.
In an interview with Travel Noire, Reeves discussed what inspired him to start the running club and his plans to expand:
Travel Noire: What hat inspired you to launch Fugee Run?
Durand Reeves: So I came over to Egypt about a year ago to lay the groundwork out for my business. And within me traveling around Egypt, I’ve noticed there’s been a void of seeing a lot of Black faces on advertising, any type of marketing, or even having groups specifically for that demographic. There’s a huge refugee community and also outside of that, a huge Black-African presence here but it just wasn’t a lot of representation.
I wanted to make space and create a platform that was, marketing specifically to our demographic and have something that they can relate to.
Travel Noire: Why, why did you feel like it was needed in Egypt?
Durand Reeves: There’s a ton of athletic groups here, but when you look at the marketing or look at who is participating in those groups, I didn’t see a lot of Black African presence there.
After doing some research, I found some people don’t have either the finances to be involved in some of those seven programs, they don’t have the time or availability because they’re working, or they just didn’t feel welcome in those spaces.
And it’s not that there’s racism, but we don’t see a lot of faces that look like ours which makes you a little hesitant to sometimes jump in.
Travel Noire: What’s next for you all?
Durand Reeves: We actually just got some interest from a group in Alexandria, Egypt.
There are about 20 people up there that started their own running group and wanted to grow it up there. The movement has organically started growing.
I always knew there’s potential but I didn’t have that expectation. Now that I know there is a need and as long as everything is consistent with the branding that we’re looking in place, whereas you know community focused and you know giving people this outlet, then I’m all for expanding all over Africa.
Source – Travel Noir